Acanthosis nigricans
☆ Mune 2022 Stiftung Warentest mhedzisiro kubva kuGermany, kugutsikana kwevatengi neModelDerm kwakangodzikira zvishoma pane nekubhadharwa kwe telemedicine kubvunzana.
Zvinowanzoitika muvanhu vakafuta.
References
Acanthosis nigricans chiratidzo cheganda chemamiriro epasi. Inowanzoonekwa mumapete eganda sehuro, muhapwa, uye groin, inoratidzika semavara erima ane velvety ane mipendero isina kujeka. Chirwere ichi chinowanzobatanidzwa nechirwere cheshuga uye insulin kuramba, asi muzviitiko zvisingawanzoitiki, zvinogona kureva gomarara mukati memuviri. Inogonawo kuratidza nekuda kwekunetseka kwehomoni kana kubva pakutora mishonga chaiyo senge steroids uye mapiritsi ekudzivirira kubata pamuviri.
Acanthosis nigricans is a cutaneous manifestation of an underlying condition. It usually develops in skin folds, such as the back of the neck, axilla, and groin, where it presents as velvety hyper-pigmented patches with poorly defined borders. Acanthosis nigricans is most commonly associated with diabetes and insulin resistance, but rarely it can be a sign of internal malignancy. It can also occur with hormone disorders or with the use of certain medications like systemic glucocorticoids and oral contraceptives.
Acanthosis nigricans is a cutaneous manifestation of an underlying condition. It usually develops in skin folds, such as the back of the neck, axilla, and groin, where it presents as velvety hyper-pigmented patches with poorly defined borders. Acanthosis nigricans is most commonly associated with diabetes and insulin resistance, but rarely it can be a sign of internal malignancy. It can also occur with hormone disorders or with the use of certain medications like systemic glucocorticoids and oral contraceptives.
Acanthosis nigricans (AN) chiitiko cheganda chakajairika chakabatana nehutano hwakasiyana-siyana se insulin kuramba, chirwere cheshuga, kufutisa, mamwe magomarara, matambudziko ehomoni, uye maitiro kumishonga. Kurapa AN kunotarisa kugadzirisa matambudziko ari pasi pehutano. Pakutanga, vanachiremba vanotarisa zviratidzo zve insulin resistance syndrome, iyo inosanganisira kufutisa, high cholesterol, high blood pressure, uye type 2 chirwere cheshuga. Vanachiremba vanowanzonyora topical retinoids sesarudzo yekutanga yekurapa, iyo inogona kubatsira nekukora kweganda. Zvisinei, vangasanyatsogadzirisa kusviba kweganda. Dzimwe nzira dzekurapa (salicylic acid, podophyllin, urea, calcipotriol) dzinodawo kushandiswa kakawanda.
Acanthosis nigricans (AN) is a common dermatologic manifestation of systemic disease that is associated with insulin resistance, diabetes mellitus, obesity, internal malignancy, endocrine disorders, and drug reactions. Treatment of AN primarily focuses on resolution of the underlying disease processes causing the velvety, hyperpigmented, hyperkeratotic plaques found on the skin. Initial considerations for the AN workup include evaluating patients for insulin resistance syndrome characterized by obesity, dyslipidemia, hypertension, and diabetes mellitus type II. For cosmetic treatment, topical retinoids are considered the first-line therapy for insulin-resistant AN by modifying keratinization rate. However, topical tretinoin requires application for long durations and improves hyperkeratosis, but not hyperpigmentation. Topical salicylic acid, podophyllin, urea, and calcipotriol also require frequent application, while TCA peels may provide a faster and less time-intense burden.
Acanthosis nigricans (AN) is a common dermatologic manifestation of systemic disease that is associated with insulin resistance, diabetes mellitus, obesity, internal malignancy, endocrine disorders, and drug reactions. Treatment of AN primarily focuses on resolution of the underlying disease processes causing the velvety, hyperpigmented, hyperkeratotic plaques found on the skin. Initial considerations for the AN workup include evaluating patients for insulin resistance syndrome characterized by obesity, dyslipidemia, hypertension, and diabetes mellitus type II. For cosmetic treatment, topical retinoids are considered the first-line therapy for insulin-resistant AN by modifying keratinization rate. However, topical tretinoin requires application for long durations and improves hyperkeratosis, but not hyperpigmentation. Topical salicylic acid, podophyllin, urea, and calcipotriol also require frequent application, while TCA peels may provide a faster and less time-intense burden.
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Izvi zvinowanzoitika kune vanhu vari pasi pemakore makumi mana, vanogona kunge vakagarwa nhaka uye vanobatana nekufutisa kana endocrinopathies, senge hypothyroidism, acromegaly, polycystic ovary chirwere, insulin-resistant chirwere cheshuga kana Cushing's chirwere.
○ Kurapa ― OTC Zvinodhaka
#40% urea cream